← All clinician topics

CLINICIAN PVC & VT GUIDE · SECTION 2 OF 3

The PVC/VT patient in clinic

Assess symptoms and risk, recognize emergencies, and coordinate cardiology/EP referral.

Assess urgency before ordering a monitor

Ongoing wide-complex tachycardia, syncope with concerning features, hypotension, ischemic chest discomfort, acute heart failure, or altered mental status requires emergency evaluation. Activate EMS. Do not delay transfer to obtain a perfect tracing.

Manage an undifferentiated wide-complex tachycardia as possible VT. A patient who is awake or has a measurable BP can still have VT. Avoid empiric verapamil or diltiazem for an uncertain wide-complex rhythm.[5]

For the stable patient

  • History: symptoms, exertional relationship, syncope, prior MI/HF, family sudden death, stimulant exposure, and medication changes.
  • EKG: capture ectopy when possible; review QRS morphology, QTc, ischemic changes, and conduction disease.
  • Monitoring: quantify PVC burden, morphologies, NSVT episodes, and symptom correlation. Review strips and recording duration.
  • Targeted tests: potassium, magnesium, renal function, and other labs guided by context.
[3]

Coordinate cardiac evaluation: echocardiography assesses function; ischemia testing and cardiac MRI are selected according to symptoms, risk, and suspected substrate. A normal echo alone does not establish that an atypical ventricular arrhythmia is benign.[4]

When to refer

Ongoing VT, instability, or repeated ICD shocks

Emergency assessment.

NSVT with syncope, reduced EF, known scar, or concerning family history

Expedited cardiology/EP evaluation; emergency disposition if current clinical risk warrants.

Frequent PVCs, impaired EF, significant symptoms, or PVCs limiting CRT pacing

Cardiology/EP referral for substrate assessment and suppression options.

Occasional PVCs with reassuring evaluation

Explain findings, address triggers, and plan follow-up.

This referral framework applies the risk and treatment principles in the cited guidance; timing remains individualized.[3][4]

Two clinic pitfalls

Apparent bradycardia: bigeminy can create a pulse deficit. Compare the electrical rate on EKG with the palpable pulse before attributing the finding to sinus-node disease.[9]

“Only a short run”: NSVT needs clinical context. Send the actual EKG/monitor strips, symptoms, burden, EF, medications, and relevant labs with the referral.